I’m not going to look for references right now, but there is current research into the anti-inflammatory effects of classical psychedelics.
Anecdotally, it has been speculated by researchers that it is in fact this effect that produces many of the observed benefits, apart from the psychedelic experience itself.
And so people are looking at analogs that can have some of the same receptor affinity profiles without causing psychedelic effects, or with much diminished psychedelic effects.
Personally I think a protocol such as what you suggest is a better approach and would work.
I had a friend who had a historical condition caused by a spinal trauma in her childhood, while professionally practicing ballet.
It would be externalised as random pains associated with her back.
She went through several prescription painkillers when her doctor told her to try sub-recreational doses of shrooms to treat the pain (obviously not prescribed).
It worked well until I believe she tripped hard, which I suppose happens with street-quality drugs and specifically with mushrooms which if not extracted have a relatively large variablity in concentrations between mushrooms bodies.
So here's both the lesson of why she stopped doing them and the obvious fact of some doctors actually knowing of the effects; but they can not advise since there no specific research on this could be used as a medication this and whether that could even be regularly prescribed as a medicine.
Anecdotally, it has been speculated by researchers that it is in fact this effect that produces many of the observed benefits, apart from the psychedelic experience itself.
And so people are looking at analogs that can have some of the same receptor affinity profiles without causing psychedelic effects, or with much diminished psychedelic effects.
Personally I think a protocol such as what you suggest is a better approach and would work.